首页|期刊导航|中风与神经疾病杂志|基底节区脑出血破入脑室合并迟发性动眼神经麻痹1例报告并文献复习

基底节区脑出血破入脑室合并迟发性动眼神经麻痹1例报告并文献复习OA

Basal ganglia hemorrhage breaking into the ventricles complicated by delayed oculomotor nerve palsy:A case report and literature review

中文摘要英文摘要

本文报告1例基底节区脑出血破入脑室后出现迟发性动眼神经麻痹的病例,探讨其临床特点及可能的发病机制,为临床诊疗提供参考.回顾性分析1例57岁男性脑出血患者的临床资料,包括病史、体格检查、影像学检查及诊疗过程,并结合国内外文献对可能的发病机制进行分析.患者头部CTA未见动脉瘤征象,治疗后患者脑出血吸收,出院后2个月眼睑下垂及眼球运动完全恢复正常.脑出血破入脑室后可能出现迟发性动眼神经麻痹,其发病机制可能与脑室内血块机化收缩压迫神经、血管痉挛等因素相关,保守治疗可能预后良好.本病例可为临床诊疗提供参考.

This article reports a case of delayed oculomotor nerve palsy after basal ganglia hemorrhage breaking into the ventricles and discusses its clinical features and possible pathogenic mechanisms,so as to provide a reference for clini-cal diagnosis and treatment.A retrospective analysis was performed for the clinical data of a male patient with intracere-bral hemorrhage aged 57 years,including medical history,physical examination,imaging examination,diagnosis,and treatment,and the possible pathogenic mechanisms were analyzed with reference to related articles in China and globally.Head computed tomography angiography showed no signs of aneurysm.The patient achieved absorption of intracerebral hemorrhage after treatment and complete resolution of blepharoptosis and recovery of eye movement at 2 months after dis-charge.Delayed oculomotor nerve palsy may occur after intracerebral hemorrhage breaking into the ventricles,and its pathogenesis may be associated with factors such as nerve compression caused by organization and contraction of intraven-tricular blood clots and vasospasm.Conservative treatment may help to achieve a good prognosis.This case can provide a reference for clinical practice.

韩雅婷;何洋;于垚;郭淮莲

北京大学人民医院神经内科,北京 100044北京大学人民医院神经内科,北京 100044北京大学人民医院神经内科,北京 100044北京大学人民医院神经内科,北京 100044

医药卫生

脑出血动眼神经麻痹头痛

Intracerebral hemorrhageOculomotor nerve palsyHeadache

《中风与神经疾病杂志》 2026 (8)

675-678,4

国家自然科学基金重点项目(81230023)

10.19845/j.cnki.zfysjjbzz.2026.0117

评论