Increased oxygen demand during exercise as a stimulus for neuroprotection:A working hypothesisOA
Increased oxygen demand during exercise as a stimulus for neuroprotection:A working hypothesis
Johannes Burtscher;Katharina Hüfner;Martin Kopp;Robert Motl;Erich Hohenauer;Luis Santos;Atbin Djamshidian;Hannelore Ehrenreich;Florian Krismer;Klaus Berek;Martin Burtscher
Department of Sport Science,University of Innsbruck,Innsbruck,Austria||Department of Psychiatry,Psychotherapy,Psychosomatics and Medical Psychology,University Hospital for Psychiatry Ⅱ,Medical University of Innsbruck,Innsbruck,AustriaDepartment of Psychiatry,Psychotherapy,Psychosomatics and Medical Psychology,University Hospital for Psychiatry Ⅱ,Medical University of Innsbruck,Innsbruck,AustriaDepartment of Sport Science,University of Innsbruck,Innsbruck,AustriaDepartment of Kinesiology and Nutrition,University of Illinois Chicago,Chicago,IL,USARehabilitation and Exercise Science Laboratory,Department of Business Economics,Health and Social Care,University of Applied Sciences and Arts of Southern Switzerland,Landquart,Switzerland||Department of Neurosciences and Movement Science,University of Fribourg,Fribourg,SwitzerlandDepartmento de Ciencias de la Educación,Universidad de Oviedo,Oviedo,SpainDepartment of Neurology,Medical University of Innsbruck,Innsbruck,AustriaExperimental Medicine,Central Institute of Mental Health,Medical Faculty Mannheim,Heidelberg University,Mannheim,GermanyDepartment of Neurology,Medical University of Innsbruck,Innsbruck,AustriaDepartment of Neurology,Medical University of Innsbruck,Innsbruck,AustriaDepartment of Sport Science,University of Innsbruck,Innsbruck,Austria
cardiorespiratory fitnessexercisehypoxia inducible factormental healthmitochondrianeurodegenerationneurological disordersneuroprotectionoxygenphysical activity
cardiorespiratory fitnessexercisehypoxia inducible factormental healthmitochondrianeurodegenerationneurological disordersneuroprotectionoxygenphysical activity
《中国神经再生研究(英文版)》 2026 (10)
4523-4528,6
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